Call for Abstracts (Poster Presentations) Stay tuned for an announcement for abstact submission deadlines for NeuroSports2023!
Rules to Follow: 1) the Abstract must be original unpublished data. You will be notified of acceptance or rejection within 7 days or less after we receive your Abstract. We accept original investigations only. 2) You must be present at your Posteron Friday February 17th) You can submit multiple posters as long as the PI (or one of his/her students/assistants) is present at the Poster.
Deadline: January 13 2023
Please Note: You still need to pay the conference registration fee if you are presenting a poster; this includes individuals who are also exhibiting at the event. This fee is waived only for those who give oral presentations. Please follow the format for Abstracts (see below).
All expenses are to be covered by the presenting author(s).
Please limit the word count to 500 words or less. This refers to the body of the text.
Poster must be written and presented in lucid English.
If you have any conflicts of interest, please denote that in the acknowledgements.
Please email your abstract to neurosportsresearch@gmail.com
Example - Abstract format
You may be fit but your genes want you fat: the FTO (fat mass and obesity-associated) gene polymorphism influences fat mass in exercise-trained individuals
Jose Antonio1, Sarah Knafo2, Ritishka Kapoor2, Jaime L. Tartar2
1Department of Health and Human Performance, Nova Southeastern University, Davie, FL, USA; 2Department of Psychology and Neuroscience, Nova Southeastern University, Davie, FL, USA
*Corresponding author - Jose Antonio PhD
Email: email here
Abstract
Background
A single nucleotide polymorphism (SNP) in the fat mass and obesity-associated (FTO) gene is a strong predictor of obesity in humans. The FTO SNP (rs1421085) results in a T to C nucleotide substitution that may result in an increased risk for obesity in individuals who carry at least one C allele. The highest expression levels of the FTO enzyme are found in arcuate nucleus of the hypothalamus, which plays a major role in appetite and eating behavior. Therefore, the purpose of this investigation was to characterize the FTO genotype in a cohort of exercise-trained men and women.
Materials and Methods
We tested 108 exercise-trained individuals that included professional mixed martial arts fighters, competitive distance runners, collegiate swimmers, stand-up paddlers as well as a cohort of recreational bodybuilders. Body composition was assessed via the dual-energy x-ray absorptiometry (DXA). Saliva samples were collected in order to genotype participants and quantify cortisol levels.
Results
The physical characteristics of the subjects were as follows (mean±SD): body weight 74.5±15.6 kg; bone mineral content 2.8±0.7 kg; fat mass 15.7±5.5 kg; lean body mass 55.9±14.4 kg; % body fat 21.6±7.0. C allele carriers had significantly higher fat mass t(107)=3.13, p < 0.01 and body fat percentage t(107)=2.68, p < 0.01, relative to the TT group (i.e., fat mass: C/- 17.3 ±5.6 kg, TT 14.2±4.6 kg; body fat percentage: C/- group 23.4±7.4 %, TT group 19.9±6.2). No other measures of body composition were associated with the FTO genotype (i.e., body mineral density, bone mineral content, or lean body mass). Moreover, cortisol levels were significantly higher in the TT group relative to the C allele carriers t(106) = 2.37, p = 0.02 (i.e., TT 0.35 ±0.35 µg/dL, C/- 0.22±0.16 µg/dL).
Conclusions
Our findings suggest that despite regular exercise training, C allele carriers on the FTO gene are still predisposed to a higher fat mass and body fat percentage. We further show that increased cortisol is not a likely pathway in which C allele carriers have a greater fat mass.
Conflict of Interest Declaration: None to declare
